Racial Variation in the Control of Diabetes Among Elderly Medicare Managed Care BeneficiariesIdentification of a ß-Cell-Specific HLA Class I Restricted Epitope in Type 1 Diabetes
Intermittent High Glucose Enhances Apoptosis Related to Oxidative Stress in Human Umbilical Vein Endothelial Cells: The Role of Protein Kinase C and NAD(P)H-Oxidase ActivationIntermittent High Glucose Enhances Apoptosis Related to Oxidative Stress in Human Umbilical Vein Endothelial Cells: The Role of Protein Kinase C and NAD(P)H-Oxidase Activation
Adverse Effects of Dietary Glycotoxins on Wound Healing in Genetically Diabetic MiceAdverse Effects of Dietary Glycotoxins on Wound Healing in Genetically Diabetic Mice
Influence of Type 2 Diabetes on Functional and Structural Properties of Coronary Artery Bypass ConduitsInfluence of Type 2 Diabetes on Functional and Structural Properties of Coronary Artery Bypass Conduits
Leptin and Wound Inflammation in Diabetic ob/ob Mice: Differential Regulation of Neutrophil and Macrophage Influx and a Potential Role for the Scab as a Sink for Inflammatory Cells and MediatorsLeptin and Wound Inflammation in Diabetic ob/ob Mice: Differential Regulation of Neutrophil and Macrophage Influx and a Potential Role for the Scab as a Sink for Inflammatory Cells and Mediators
Effect of Type 1 Diabetes on the Gender Difference in Coronary Artery Calcification: a Role for Insulin Resistance?: The Coronary Artery Calcification in Type 1 Diabetes (CACTI) StudyEffect of Type 1 Diabetes on the Gender Difference in Coronary Artery Calcification: a Role for Insulin Resistance?: The Coronary Artery Calcification in Type 1 Diabetes (CACTI) Study
Parathyroid Hormone–Related Protein Induces Insulin Expression Through Activation of MAP Kinase–Specific Phosphatase-1 That Dephosphorylates c-Jun NH2-Terminal Kinase in Pancreatic ß-CellsParathyroid Hormone–Related Protein Induces Insulin Expression Through Activation of MAP Kinase–Specific Phosphatase-1 That Dephosphorylates c-Jun NH2-Terminal Kinase in Pancreatic ß-Cells
Rapamycin Has a Deleterious Effect on MIN-6 Cells and Rat and Human IsletsRapamycin Has a Deleterious Effect on MIN-6 Cells and Rat and Human Islets
Coordinated Upregulation of Oxidative Pathways and Downregulation of Lipid Biosynthesis Underlie Obesity Resistance in Perilipin Knockout Mice: A Microarray Gene Expression ProfileCoordinated Upregulation of Oxidative Pathways and Downregulation of Lipid Biosynthesis Underlie Obesity Resistance in Perilipin Knockout Mice: A Microarray Gene Expression Profile
Transgenic Neuronal Expression of Proopiomelanocortin Attenuates Hyperphagic Response to Fasting and Reverses Metabolic Impairments in Leptin-Deficient Obese MiceTransgenic Neuronal Expression of Proopiomelanocortin Attenuates Hyperphagic Response to Fasting and Reverses Metabolic Impairments in Leptin-Deficient Obese Mice
Learned Meal Initiation Attenuates the Anorexic Effects of the Melanocortin Agonist MTIILearned Meal Initiation Attenuates the Anorexic Effects of the Melanocortin Agonist MTII
Linkage Analysis of a Composite Factor for the Multiple Metabolic Syndrome: The National Heart, Lung, and Blood Institute Family Heart StudyLinkage Analysis of a Composite Factor for the Multiple Metabolic Syndrome: The National Heart, Lung, and Blood Institute Family Heart Study
Association of Solute Carrier Family 12 (Sodium/Chloride) Member 3 With Diabetic Nephropathy, Identified by Genome-Wide Analyses of Single Nucleotide PolymorphismsAssociation of Solute Carrier Family 12 (Sodium/Chloride) Member 3 With Diabetic Nephropathy, Identified by Genome-Wide Analyses of Single Nucleotide Polymorphisms
Permanent Neonatal Diabetes Caused by Glucokinase Deficiency: Inborn Error of the Glucose-Insulin Signaling PathwayPermanent Neonatal Diabetes Caused by Glucokinase Deficiency: Inborn Error of the Glucose-Insulin Signaling Pathway
Recombinant Fabs of Human Monoclonal Antibodies Specific to the Middle Epitope of GAD65 Inhibit Type 1 Diabetes–Specific GAD65AbsRecombinant Fabs of Human Monoclonal Antibodies Specific to the Middle Epitope of GAD65 Inhibit Type 1 Diabetes–Specific GAD65Abs
Target Cell Expression of Suppressor of Cytokine Signaling-1 Prevents Diabetes in the NOD MouseTarget Cell Expression of Suppressor of Cytokine Signaling-1 Prevents Diabetes in the NOD Mouse
Discovery of Gene Networks Regulating Cytokine-Induced Dysfunction and Apoptosis in Insulin-Producing INS-1 CellsDiscovery of Gene Networks Regulating Cytokine-Induced Dysfunction and Apoptosis in Insulin-Producing INS-1 Cells
Identification of Subjects with Insulin Resistance and ß-Cell Dysfunction Using Alternative Definitions of the Metabolic SyndromeIdentification of Subjects with Insulin Resistance and ß-Cell Dysfunction Using Alternative Definitions of the Metabolic Syndrome
Contribution of Elevated Free Fatty Acid Levels to the Lack of Glucose Effectiveness in Type 2 DiabetesContribution of Elevated Free Fatty Acid Levels to the Lack of Glucose Effectiveness in Type 2 Diabetes
Prevalence and Clinicopathological Characteristics of Islet Amyloid in Chinese Patients With Type 2 DiabetesPrevalence and Clinicopathological Characteristics of Islet Amyloid in Chinese Patients With Type 2 Diabetes
Extracellular Glucose in Rat Ventromedial Hypothalamus During Acute and Recurrent HypoglycemiaExtracellular Glucose in Rat Ventromedial Hypothalamus During Acute and Recurrent Hypoglycemia
Counterregulatory Hormone and Symptom Responses to Insulin-Induced Hypoglycemia in the Postprandial State in HumansCounterregulatory Hormone and Symptom Responses to Insulin-Induced Hypoglycemia in the Postprandial State in Humans
Chronic Exposure to Interleukin-6 Causes Hepatic Insulin Resistance in MiceChronic Exposure to Interleukin-6 Causes Hepatic Insulin Resistance in Mice
Timing of Changes in Interstitial and Venous Blood Glucose Measured With a Continuous Subcutaneous Glucose SensorTiming of Changes in Interstitial and Venous Blood Glucose Measured With a Continuous Subcutaneous Glucose Sensor
Hypothesis: Shifting the Equilibrium From Activity to Food Leads to Autonomic Unbalance and the Metabolic SyndromeHypothesis: Shifting the Equilibrium From Activity to Food Leads to Autonomic Unbalance and the Metabolic Syndrome
Increased Insulin Sensitivity and Hypoinsulinemia in APS Knockout MiceIncreased Insulin Sensitivity and Hypoinsulinemia in APS Knockout Mice
Director Editorial
